I wouldn’t put it this way for research, as science isn’t about winning. But I also returned last week from China with similar sentiments about Chinese prowess.
www.nytimes.com/2026/02/10/o...
Happy to share a cartoon-based illustration of our recent findings of disease resistance gene ZmRRS1/ZmMDH7 in maize (doi.org/10.1002/advs...). The pictures can tell the story, I hope.
1/2 What's best: a field-first or lab-first approach? No easy answers but differences between lab and field should not be seen as failure but motivate further inquiry and allow complementary discovery. Read our thoughts on this here:
www.sciencedirect.com/science/arti...
Recommended 10/10! Thanvi is everything you’re looking for in an advisor!
Out First Release in @science.org
A pollen ligand, SIPS, binds to the female receptor, SRK. SIPS binds to a conserved region of SRK, for interspecific incompatibility, unlike the self-incompatibility factor, which binds to a different, variable region
www.science.org/doi/10.1126/...
#PlantScience
We (Nordborg & Weigel labs) need input on the next generation of genome browsers & data download modes for the #Arabidopsis #1001GenomesPlus project. We have now a curated collection of over 500 long read genomes.
Please help us by filling out this questionnaire: docs.google.com/forms/d/e/1F...
We established a simple protocol for reliable, high-throughput infection with R. solani in the field. This helps us identify maize malate dehydrogenase as a new resistance gene by enhancing mtROS accumulation. Newly published in
@AdvancedScience
: doi.org/10.1002/advs....
Thanvi has great enthusiasm and motivation for Science. Congratulations, Thanvi~
Congratulations, Thanvi~
Now with a Research Briefing: "Glycosylation disruption is a new virulence strategy for a plant fungal pathogen" rdcu.be/eHeag
Fusarium secretes an apoplastic effector that disrupts N-glycosylation of the immune receptor ZmLecRK1, leading to its degradation.
New Letter: "An apoplastic fungal effector disrupts N-glycosylation of ZmLecRK1, inducing its degradation to suppress disease resistance in maize" rdcu.be/eGD4E
A fungal effector promotes a cell-surface receptor autophagy-mediated degradation to dampen plant immunity.
Research Briefing in @natplants.nature.com
"Glycosylation disruption is a new virulence strategy for a plant fungal pathogen": highlight of our recent findings published in Nature Plants doi.org/10.1038/s414....
Out in @NaturePlants. We discover that the fungal pathogen Fusarium graminearum secretes an apoplastic effector that disrupts the N-glycosylation of a maize immune receptor, thereby inducing its degradation via selective autophagy. Very proud of the team~ doi.org/10.1038/s414...
Out after peer review, collaborative study from Nordborg & Weigel labs with help from many others. Not the largest collection of new Arabidopsis thaliana genomes, but we hopefully put forward some good ideas for how to think about pangenomes and their analysis!
www.nature.com/articles/s41...
This work started nearly 10 years ago and was once my main postdoctoral project at @plantevolution.bsky.social before I slowed work on it to a trickle because it became confusing. But it always remained extremely interesting.
journals.plos.org/plosbiology/...
Super excited to share our new preprint! 🎉 This project has been an incredible learning experience, thanks to the amazing lab and wonderful people I get to work with. Huge thanks to my supervisor! @plantevolution.bsky.social
